Assistant Professor,Institute of Medical Science.
Effects of the differential expression of ZO-1 and ZO-2 on podocyte structure and function.
Forced expression of vascular endothelial growth factor-A in podocytes decreases mesangial cell numbers and attenuates endothelial cell differentiation in the mouse glomerulus.
The reduction of heparan sulphate in the glomerular basement membrane does not augment urinary albumin excretion.
Tubulointerstitial fibrosis can sensitize the kidney to subsequent glomerular injury.
Lipophagy maintains energy homeostasis in the kidney proximal tubule during prolonged starvation.
High-Fat Diet-Induced Lysosomal Dysfunction and Impaired Autophagic Flux Contribute to Lipotoxicity in the Kidney.
Autophagy Inhibits the Accumulation of Advanced Glycation End Products by Promoting Lysosomal Biogenesis and Function in the Kidney Proximal Tubules.
Erratum: Distinct neural mechanisms for the control of thirst and salt appetite in the subfornical organ.
Natriuretic peptide receptor guanylyl cyclase-A pathway counteracts glomerular injury evoked by aldosterone through p38 mitogen-activated protein kinase inhibition.
Foxc1 and Foxc2 are necessary to maintain glomerular podocytes.
Distinct neural mechanisms for the control of thirst and salt appetite in the subfornical organ.
Crucial Role of Mesangial Cell-derived Connective Tissue Growth Factor in a Mouse Model of Anti-Glomerular Basement Membrane Glomerulonephritis.
3D Culture Supports Long-Term Expansion of Mouse and Human Nephrogenic Progenitors.
Adipocytes do not significantly contribute to plasma angiotensinogen.