claim: convalescent covid-19 patients are susceptible to endothelial dysfunction due to persistent immune activation
evidence_1: convalescent patients had elevated circulating endothelial cells (cecs), and those with underlying cardiovascular risk had more pronounced endothelial activation hallmarks (icam1, p-selectin or cx3cl1) expressed by cecs.", "several proinflammatory and activated t lymphocyte-associated cytokines correlated positively with cec measures, implicating cytokine-driven endothelial dysfunction.")