claim: covid-19 ards is characterized by a dysregulated host response that differs from cytokine storm and may be modified by dexamethasone.
evidence_1: covid-19 ards was characterized by a dysregulated host response with increased pten signaling and elevated expression of genes with non-canonical roles in inflammation and immunity that were predicted to be modulated by dexamethasone and granulocyte colony stimulating factor.", "in contrast to a cytokine storm, we observed reduced proinflammatory gene expression in covid-19 ards when compared to ards due to other causes.", "covid-19 ards is characterized by a dysregulated host response that differs from cytokine", 
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  
  "in summary, assessment of host gene expression in the lower airways of patients with covid-19 ards did not demonstrate cytokine storm but instead revealed a unique and dysregulated host response predicted to be modified by dexamethasone.")