 This theory successfully predicts that the human genome is shorter than other Great Ape genomes and that there are significantly more defunct pseudogenes with functional homologs in the chimpanzee genome than vice versa.While the protein coding portion of the FOXP2 gene is identical to that in Neanderthals, there is one point mutation in the regulatory part thereof (modern humans having a T where Neanderthals and all nonhuman vertebrates have an A).The observation that the effect of that difference is that the modern human FOXP2 gene does not interact with RNA from other genes while all other vertebrate including Neanderthal varieties did agrees with the idea that modern human origins was marked by the elimination (not formation) of complex gene networks, as predicted by this model.