became eligible to compete as a Non-Sporting breed, effective January 1, 1984. The breed was recognized by the Fédération Cynologique Internationale in 1987 and placed in Group 9 Companion and Toy Dogs, Section 5 : Tibetan breeds. Health The Tibetan Spaniel is a generally healthy dog with a life expectancy of 13–16 years. Progressive retinal atrophy Progressive retinal atrophy is a genetic disease that can occur in the breed. The disease is an inherited form of blindness in dogs that has in two forms: generalized PRA and central PRA. Generalized PRA is primarily a photoreceptor disease and is the form found in Tibetan Spaniels. The clinical signs have been observed between 1½ and 4 years, but as late at seven years. The disease is painless and affected dogs become completely blind. Currently there is no treatment, but affected dogs generally adapt well to their progressive blindness. The earliest clinical sign of progressive retinal atrophy is "night blindness." The dog cannot see well in a dimly lit room or at dusk. The dog will show a reluctance to move from a lighted area into darker surroundings. The night blindness develops progressively into complete blindness. The British institution Animal Health Trust (AHT) devoted intensive research for PRA in Tibetan Spaniels, isolating the responsible gene. The mutation was identified by Louise Downs, as part of her PhD studies. A DNA test based on this mutation became available July 8, 2013. Responsible breeders are working hard to eliminate PRA within the breed. Currently, the International Tibetan Spaniel Working Party collects health data including PRA history. Portosystemic shunt A portosystemic shunt is an abnormal vessel that allows blood to bypass the liver, one of the body's filters, so that it is not cleansed. This rare condition in Tibetan Spaniels is often referred to as a "liver shunt". Most shunts cause recognizable symptoms by the time a dog is a young adult but are occasionally diagnosed only later in life. Since the severity of the condition can vary widely depending on how much blood flow is diverted past the liver it is possible for a lot of variation in clinical signs and time of onset. Often, this condition is recognized after a puppy fails to grow, allowing early diagnosis. Signs of portosystemic shunts include poor weight gain, sensitivity to sedatives (especially diazepam), depression, pushing the head against a solid object, seizures, weakness, salivation, vomiting, poor appetite,