varies in populations and there are no studies which have measured a correlation between chlorfenvinphos exposure and cholinesterase inhibition. There have been suggestions that chlorfenvinphos or its metabolites would be a better biomarker of exposure than its cholinesterase activity inhibition. Biomarkers of effect In combination with analysis of reductions in cholinesterase activity in the blood, symptoms of organophosphate poisoning can be used to identify victims of organophosphate poisoning. These symptoms are not specific for chlorfenvinphos, but for anticholinesterase compounds in general. Treatments of exposure Ingestion of chlorfenvinphos, either by accident or through suicidal intent, can be treated as with other acute organophosphate poisonings. This includes a combination of three approaches: Administration of an anticholinergic such as atropine, considered an antidote; Administration of a cholinesterase reactivator, in the pyridinium oxime family, usually pralidoxime; Administration of anticonvulsants, e.g. benzodiazepines (of which diazepam is most effective). The efficacy of oxime treatment is controversial. Ingestion of organophosphates as residues on food rarely reaches clinically relevant doses. References External links ATSDR en Español - ToxFAQs: Clorfenvinfos Acetylcholinesterase inhibitors Organophosphate insecticides Organochlorides Ethyl esters Chlorobenzenes