with extinction, who are able to detect a single target in any location, with a deficit only for multiple concurrent targets. Therefore, extinction can be regarded as a pathological, spatially specific exaggeration of the normal difficulty in distributing attention to multiple targets so we can predict that it should be reduced if the two competing events could be grouped together. Several recent findings from right-parietal patients with left extinction confirm this prediction, suggesting that grouping mechanisms may still operate despite the pathological spatial bias of the patient to influence whether a particular stimulus will reach the patient's awareness. Thus extinction is reduced when the concurrent target events can be linked into a single subjective object, becoming allies rather than competitors in the bid to attract attention. Furthermore, the extent of residual processing extinct stimuli can vary from one patient to another, depending on the exact extent of their lesion. The examples of residual unconscious processing so far all concern the visual modality although evidence is starting to emerge concerning similar effects for extinguished tactile and auditory stimuli. Physiology/characteristics Extinction as well as spatial neglect are deficits caused by large lesions in the vasculatory territory of the medial cerebral artery [20]. Some studies say that extinction occurs after damage to the right or left hemisphere. Patients with extinction do not report stimuli located in space contralateral to their damaged hemisphere when the stimuli are presented simultaneously with ipsilesional stimuli. Extinction to double simultaneous stimulation is not only attributed to a primary sensory deficit since these patients are aware of contralateral stimuli presented individually. Attentional Deficit Hyperactivity Disorder (ADHD) is more focused on as a role of abnormal sensory processing of contralesional input. The patients have a pathologically limited attentional capacity and an attentional bias towards ipsilesional space, they are more likely to attend to and become aware of ipsilesional stimuli at the expense of contralesional ones. It is noteworthy that the right Temporoparietal Junction (TPJ) has been linked to a number of cognitive functions that suggest a role in modulating competitive interactions between stimulus representations, which would converge with the importance of this area for the attentional deficit displayed by extinction patients. The critical lesion site responsible for the syndrome has been debated for more than a decade. Different criterion was used to identify extinction in their patient samples, which lead to inconsistencies in the critical lesion sites reported across studies.