to the renal tissues caused by ADR were decreased by GATE suggesting its protective effect in ADR-induced renal injury. In this study, network pharmacology was used to better comprehend the connections between Nephrotic Syndromes treatment and the administration of GATE. The results observed from network pharmacology discovered that multiple pathways and targets were involved in the modification of GATE on Nephrotic Syndromes (NS). The expected pathways and targets of GATE involved in Nephrotic Syndromes were tightly related with inflammation and oxidative stress. In this study conducted by Lihua Tan, it was further observed that the ADR injected mice had relatively higher intracellular ROS (Reactive oxygen species) production in their renal tissue, while the GATE treatment substantially reversed this alteration caused by ADR. In contrast to the ADR injected mice, the GATE treated mice had reduced MDA (Malonaldehyde) levels. In the ADR injected mice, the SOD (Superoxide dismutase) activity was reduced in renal tissues and plasma and was identified to be under Nephrotic Syndromes condition, which was observed to be repaired by the GATE treatment. The results from this study, which were consistent with network pharmacology, suggested that the GATE treatment could defend mice from renal injury by mitigating ADR-induced oxidative stress and inflammation in Nephrotic Syndromes (NS) mice. Taxonomy The following table displays the taxonomic report of Glycine tabacina. References External links Glycine tabacina. PlantNet. Retrieved 25 September 2009. tabacina Flora of New South Wales Flora of Queensland Flora of Victoria (Australia) Flora of Western Australia Taxa named by George Bentham Taxa named by Jacques Labillardière Fabales of Asia Fabales of Australia