that led to the cytotoxicity in U87 and A172 glioblastoma cells. The first method showed CTAB exchanging with phospholipids causing rearrangement of the membrane allowing β-galactoside to enter into the cell by way of cavities. At low concentrations, there are not enough cavities to cause death to the cells, but with increasing the CTAB concentration, more phospholipids are displaced causing more cavities in the membrane leading to cell death. The second proposed method is based on the dissociation of CTAB into CTA+ and Br− within the mitochondrial membrane. The positively charged CTA+ binds to the ATP synthase not allowing H+ to bind stopping the synthesis of ATP and resulting in cell death. See also Cetrimonium chloride - The corresponding chloride salt Behentrimonium chloride - A C25 structural analogue References Further reading Merck Index, 11th Edition, 1989. Drug information Antiseptics Household chemicals Quaternary ammonium compounds Cationic surfactants Bromides