We note that in both 3-YbEu and 3-DyYb, while excitation of the hfac ligands results in emission from the Ln 3+ coordinated to the hfac ligands in the N,Nʹ binding site (ET pathway I), minor emission contributions were always observed from the Ln 3+ coordinated to the O,Oʹ binding site. This is consistent with a less efficient, long-range ET pathway (eg. Förster Resonance ET) operating in parallel with short-range pathway I. Alternatively, it is possible that this arises from excitation of a high-energy transition of pd •-, which features some absorption in the same region as the hfac ligands. However, population of the lower-lying excited states of pd •-(visible excitation) does not result in emission from Dy 3+ in 3-DyYb, whereas higher energy (UV excitation) does. Thus, either ET may occur from a higher excited state of pd accessed by UV irradiation (anti-Kasha type behaviour) or long-range ET from distant hfac ligands is operative and competing with pathway I. Our current data does not distinguish these pathways.