Recently, mitochondrial function has become a major focus of study in relation to Alzheimer's disease. Mitochondria, subcellular organelles responsible for ATP production, are vital to neurons due to their high energy demands, and low ATP levels indicate cell death. Mitochondrial fusion and fission occur continually but can result in irreparable loss when mutations occur in proteins that mediate these processes. Several proteins are involved in mitochondrial dynamics, including Optic Atrophy-1, Dynamin-Related Protein-1 (DLP-1), Mitochondrial Fission 1, Mitofusin-1, and Mitofusin-2. Optic Atrophy-1 is found in the inner-space membrane and mediates fusion processes, while DLP-1 is in the interface of the mitochondrial membrane and mediates fission processes.