A system involving numerous proteins and cells such as neutrophils, macrophages, killer T cells, and various cytokines has been established in the immune system of the living body to contribute to infection defense. Neutrophils and macrophages, the center of the immune system, phagocytose foreign pathogenic viruses that have infiltrated the body and use of reactive oxygen species (ROS) to oxidize and decompose them. Thus, superoxide radical anion (O2 •-), the center of the ROS family in the terminal of the immune mechanism, is produced and oxidatively decomposes foreign substances such as viruses. However, it is said that when excessively produced O2 •-attack cells, it induces tissue inflammation. Therefore, immune oxidation must be used to break out the virus while also keeping it from multiplying, and an analog drug that acts under inflammatory conditions is required. In an inflamed organ, the degradation and inactivation of analog drugs due to the ROS redox reaction under the immune system are inevitable. Therefore, it is important to know the redox reaction between ROS and antiviral drugs in preventing its degradation and maximizing the concentration at the inflamed organ.