Beyond the immediate comparison with analytical solutions, our numerical framework illuminates how local geometric features of the intracellular landscape can drive emergent behaviors. In realistic cell environments, organelles, cytoskeletal filaments, and other compartments can create spatially complex barriers or partial obstructions to substrate diffusion, effectively sculpting chemical gradients. Our results suggest that even modest blocker sizes, when strategically located relative to a substrate source, can substantially redirect flow lines, leading to local "hot spots" or "shadows" of substrate concentration. This altered distribution may enable otherwise passive compartments to exhibit directed motion or accumulation patterns purely by virtue of how they reshape the local environment. Consequently, such findings have broad relevance for understanding how non-motor transport pathways might coordinate macromolecular organization inside cells or within synthetic vesicular systems, highlighting the potential for controlling microscale transport by simply introducing carefully placed blockers or tuned consumption rates. Interestingly, our theoretical findings-in which the introduction of enzymatic consumption generates new stagnation points that can promote localized clustering-stand in apparent contrast to experimental observations by Steinkühler et al . In their work, BMCs with active enzymatic activity actually reduced large-scale aggregations, rather than causing them. This discrepancy suggests that while our model accurately captures how local gradients are altered by consumption, it may not include other important effects that can disrupt or prevent clustering in a real cellular environment. Possibilities include hydrodynamic coupling, additional regulatory factors, or highly dynamic substrate turnover rates that differ from the simplified steady-state flux used here.