Elucidated gene targets of miRNAs related to LC likely have a clear relation to cancer. For example, Figure shows how one of selected target genes, RPS6KA5 triggers cancer proliferation. RPS6KA5 (MSK1), which is shown in the figure. This is an area of future study because it shows that the RPS6KA5 target gene of miRNA and the pathway it operates in promotes cancer growth, which is the central carbon metabolism in cancer, or hsa05230. We found that this pathway was more linked with laryngeal cancer-associated miRNA, so we can look further into these pathways for treating cancer in the future. Thus, the presence of this target gene and its pathway give an indication of cancer presence and are also a potential target for controlling cancer growth. Similarly, the HT-based model identified the MDM1 target gene of miRNA to be associated with LC. This gene deactivates p53, which causes decreased cancer proliferation. This gene's interaction can be visualized in the hsa05206 pathway, which is for microRNAs in cancer. In addition, we found that the TET2 target gene of miRNA was less expressed in LC-associated miRNA, indicating that LC-associated miRNAs may downregulate TET2. Downregulation of TET2 can prevent its ability to suppress tumors, which is an important area to investigate for cancer proliferation.