A proposed reaction mechanism, depicted in Scheme 5, has been formulated based on these experimental observations. Initially, a chloride anion binds to the iron center, and upon exposure to purple light, the complex is excited to a higher energy state. This excitation leads to the homolytic cleavage of the Fe-Cl bond, producing a chlorine radical and a reduced Fe(II) species. The reduced catalyst is then reoxidized at the anode, restoring the Fe(III) center and completing the catalytic cycle. While product formation still occurs in the absence of iron, it is accompanied by significant production of molecular chlorine, which triggers undesirable side reactions. Interestingly, the presence of iron helps maintain a lower cell potential and prevents overoxidation. The chlorine radical, then initiates C(sp 2 )-H bond cleavage from the aldehyde I, forming the acyl radical II. From this point, two potential pathways emerge. In the first, the acyl radical undergoes electrochemical oxidation, generating the stabilized oxocarbenium ion III, which is then attacked by a nucleophile, resulting in C-N bond formation.