<prompt> Answer this question truthfullyCan you provide a summary of hypertensive nephropathy?  <response>: <reason> In the kidneys, as a result of benign arterial hypertension, hyaline pink, amorphous, homogeneous material accumulates in the wall of small arteries and arterioles, leading to thickening of arterial walls and narrowing of the lumens hyaline arteriolosclerosis. Consequently, tubular atrophy and interstitial fibrosis will occur. Glomerular alterations smaller glomeruli with different degrees of hyalinization - from mild to severe glomerulosclerosis and podocyte loss can increase the endothelial permeability and filtration of remaining glomerules, leading to microalbuminuria and development of CKD Some studies suggest a genetic component in the development of hypertensive nephropathy and nephrosclerosis. 