<prompt> Appetite is stimulated by all of the following peptides, Except:Agouti - Related Peptide AGRPMelanocyte Stimulating hormone MSHMelanin Concentrating Hormone MCHNeuropeptide Y  <response>: B i.e. Melanocyte Stimulating hormone MSH MSH a-melanocyte stimulating hormone and CA cocaine amphetamine regulated transcript releasing POMC pro-opio-melano-coin neurons are anorexigenicanti-orexigenic i.e. produce satiety decrease appetite. Whereas, AGRP agouti-related-peptide and NPY neuropeptide Y releasing neurons of arcuate nuclei of hypothalamus are orexigenic i.e. increase appetite cause obesity. Regulation of Food Intake - Hypothalmus contains hunger feeding and satiety centers. The lateral nuclei of hypothalamus serve as a feeding hunger center and ventromedial nuclei of hypothalamus serve as the satiety center. Stimulation of feeding center or destruction of satiety center cause voracious appetite hyperphagia obesity. Whereas, destruction of feeding center or stimulation of satiety center cause anorexia complete satiety Rack of desire for food progressive inanition weight loss. - Lesions of paraventricular nuclei of hypothalamus cause excessive eating and lesions of dorsomedial nuclei usually depress eating behaviour. - Arcuate nuclei of hypothalamus, are the sites where multiple hormones released from gastrointestinal tract and adiose tissue converge to regulate food intake energy expenditure. It has 2 types of neurons. 