<prompt> High cardiac output low peripheral resistance shockHypovolemic shockCardiogenic shockSeptic shockAnaphylactic Shock  <response>: C Septic shock SEPTIC SHOCK: An inflammatory stimulus eg, a bacterial toxin triggers production of proinflammatory mediators, including I NF and IL-1. These cytokines cause neutrophil-endothelial cell adhesion, activate the clotting mechanism, and generate microthrombi Initially, arteries and arterioles dilate, decreasing peripheral arterial resistance; cardiac output typically increases. This stage has been referred to as warm shock. Later, cardiac output may decrease, BP falls with or without an increase in peripheral resistance, and typical features of shock appear. With sepsis, patients typically have fever, tachycardia, diaphoresis, and tachypnea; BP remains normal. Other signs of the causative infection may be present. 