<prompt> Bleeding time is increased in all except:ThrombocytopeniaThrombastheniaRenal failureAcquired hemophilia  <response>: aPTT is increased in acquired hemophilia Glanzmanns Thrombasthenia: AR, platelet lack receptor of Gp IIbIIIa for fibrinogen, which form the bridges between platelets during aggregation. Glanzmanns thrombasthenia is a defect of platelet functionaggregation. It presents as superficial mucocutaneous bleeding with normal platelet count and single isolated platelets without clumping on peripheral smear. Platelet numbers and morphology are normal, but the bleeding time is markedly prolonged. Platelet dysfunction in renal failure is due retention of uremic toxins, anemia,and nitric oxide. 