<prompt> All are seen in Brown - Sequard syndrome exceptIL Pyrimidal tract featuresCL Dorsal columnCL Spinothalamic tractIL Plantar extensor  <response>: C i.e. Release norepinephrineTyramine, ephedrine amphetamine TEA augment autonomic activity by releasing norepinephrineQ from endings of post-ganglionic neurons. Stnne drugs and toxins affecting Autonomic activity IPSite of ActionCompounds That Augment Autonomic ActivityCompounds That Depress Autonomic ActivitySympathetic andparasympathetic ganglia Stimulate postganglionic neurons- Nicotine- Low concentrations of acetylcholine Inhibit acetylcholinesterase- DFP diisopropyl fluorophosphate- Physostigmine Eserine- Neostigmine Prostigmin- Parathion Block conduction- Hexamethonium C-6- Mecamylamine inversine- Pentolinium- Trimethaphan Arfonad- High concentration of acetylcholineEndings ofpostganglionicnoradrenergic neurons Release norepinephrine- Tyramine- Ephedrine- Amphetamine Block norepinephrine synthesis- Metyrosine Demser Interfere with norepinephrine storage- Reserpine- Guanethidine Ismelin Prevent norepinephrine release- Bretylim Bretylol- Guanethidine Ismelin Form false transmitters- Methyldopa Aldomet Muscarinic receptors Atropine, scopolamine a adrenergic receptors Stimulate alreceptors- Methoxamine Vasoxyl- Phenylephrine Neo-Synephrine Block a receptors- Phenoxybenzamine Dibenzyline- Phentolamine Regitine- Prazosin Minipress block al- Yohimbine blocks a2 b adrenergic receptors Stimulate 13 receptors- Isoproterenol Isuprel Block receptors- Propranolol Inderal and others blocks 01- Atenolol Tnormin and others blocks 01Butoxamine blocks 02 C i.e. Release norepinephrine 