<prompt> Conjugated hyperbilirubenemia seen in:ACrigler-Najjar syndrome I IIBGilbert syndromeCDubin-Johnson syndromeDRotor syndromeEPhysiological jaundiceCDABDABC  <response>: Predominantly Unconjugated Hyperbilirubinemia Excess production of bilirubin Hemolytic anaemias Resorption of blood from internal hemorrhage e.g. alimentary tract bleeding, hematomas.Ineffective erythropoiesis syndromes e.g. pernicious anaemia, thalassemia Reduced hepatic uptake Drug interference with membrane carrier systems Some cases of Gilbert syndrome Impaired bilirubin conjugation Physiologic jaundice of the newborn decreased UGT 1A1 activity decreased excretion.Breast milk jaundice β -glucuronidases in milk Genetic deficiency of UGT 1A1 activity Crigler-Najjar syndrome types 1 and II Gilberts syndrome mixed etiologiesDiffuse hepatocellular disease e.g., viral or drug-induced hepatitis, cirrhosis PredominantlyConjugated Hyperbilirubinemia Deficiency of canalicular membrane transporters Dubin Johnsons syndrome Rotors syndrome Impaired bile flow 