<prompt> Bells palsy isHemiparesis with contralateral facial nerve palsyIpsilatera lower motor meuron lesionIpsilateral upper motor neuron lesionContralateral upper motor neuron lesions  <response>: B. i.e. Ipsilateral LMN paralysis 4- Dhingra 4th 104 - Maqbool 11th BELLS PALSY - Idiopathic infranuclear lesion of the facial nerve more common in diabetic angiopathy and pregnant women retension of fluid BELLS PHENOMENON - up and out rolling of the eyeball during forceful closureClinical presentation - LMN paralysis patients is unable to close eye, saliva dribbles from the angle of mouth, face become asymmetrical, Tears flow down from the eye epithora stapedial reflex or loss of taste Epiphora in bells palsy is due to - lacrimal pump failure Melkerssons syndrome - Triad - Facial paralysis, swelling of lips, and fissured tongue Recurrent facial palsy - causes - Melkerssons syndrome DM, sarcoidosis and tumoursPoor prognostic indicators - advanced age, hyperacusis, severe initial painBilateal facial paralysis - seen in GB syndrome sarcoidosis, sickle cell disease, acute leukemia, bulbar palsy, leprosy and sother systemic disorders.Supra nuclear central palsv primarily involves the lower part of face and emotional responses may be intact eg- the patients not be able to show you his teeth but will smile to a Joke. Nuclear palsv affects all ipsilateral muscles of facial expression resulting in paralysis of the ipsilateral side eg. mouth is pulled to the normal side and may droop on the affected side facial crease are effaced, eyelid may not close. 