<prompt> Conjugated hyperbilirubinemia is seen in -a Rotors syndb Dubin Johnson syndc Gilberts syndd Breast milk jaundicee Crigler Najjar syndromeacabadbc  <response>: Predominantly Unconjugated HyperbilirubinemiaExcess production of bilirubin Hemolytic anemiasResorption of blood from internal hemorrhage e.g.,alimentary tract bleeding, hematomasIneffective erythropoiesis syndromes e.g. permicious anemia, thalassemiaReduced hepatic uptakeDrug interference with membrane carrier systemsSome cases of Gilbert syndromeImpaired bilirubin conjugationPhysiologic jaundice of the newborn decreased UGT1A1 activity,decreased excretionBreast milk jaundice β-glucurondases in milkGenetic deficiency of UGT1A1 activity Crigler-Najjar syndrome types I and IIGilbert syndrome mixed etiologiesDiffuse hepatocellular disease e.g. viral or drug-induced hepatitis, cirrhosisPredominantly conjugated hyperbilirubinemiaDeficiency of canalicular membrane transponers Dubin-Johnson syndrome,Rotor syndromeImpaired bile flow 