<prompt> Regarding scoline apnoea all are true exceptCaused by suxamethoniumCab be inheritedDeficiency of cholinesteraseMoality is very low in recent times with proper management  <response>: C i.e. Deficiency of cholinesterase Scoline apnoea is prolonged residual paraysis after administration of succinyl choline suxamethonium 1t apnoea. It can be seen in patients with hereditary inherited atypical pseudochdinesterase enzyme homozygous heterozygousQ and phase II non depolerizing blockQ. Decreased level of normal pseudocholinestrerase not choline esterase enzymeQ would usually not result in prolonged apnea unless the surgery was very sho. In other words, in contrast to doubling or tripling of blockade duration seen in patients with heterozygous atypical enzyme or patients with low normaltypical enzyme levels, patients with homozygous atypical enzyme will have a very prolonged blockade 4-8 hrs following Sch administration. Prolonged paralysis apnoea should be treated with continued mechanical ventilation until muscle function returns to normal which has reduced moality to very low levelsQ. 