<prompt> All of the following are examples of predominantly unconjugated hyperbilirubinemia, exceptGilbes syndromeCriggler-Najjar Syndrome type IDubin-Johnson hyperbilirubinemiaCriggler-Najjar Syndrome type II  <response>: Congenital HyperbilirubinemiasCongenital hyperbilirubinemiasInheritanceBiochemical defectClinical featuresUnconjugated hyperbilirubinemiaCrigler Najjar syndrome type I ARAbsent UGT1A1 Glucronyl transferase Liver normal; Bile colourless;Kernicterus in newborn;No response to phenobarbitone Total Sr. bilirubin - 20 - 45 mgdlCrigler Najjar syndrome type II ADReduced activity of UGT1A1 0-10Liver normal; Bile pigmented;Kernicterus in newborn rare;Response to phenobarbitone Total Sr. bilirubin - 20 mgdlGilbe syndrome ADReduced activity of UGT1A1 10-33Liver normal; Bile normal colour;Kernicterus never occurs;Phenobarbitone normalizes bilirubinTotal Sr. bilirubin - 4 mgdlConjugated hyperbilirubinemiaDubin Johnson syndrome ARImpaired biliary excretion of bilirubin glucuronides Liver darkly pigmented Lysosomes contain polymers of epinephrine metabolites Rotor syndromeARDecreased hepatic uptake and storage and decreased bilirubin excretion Liver normal Refer: Harrisons Principles of Internal Medicine, 18th edition, pg no: 2532-2536 