<prompt> Metabolic alkalosis is seen inPrimary mineralocorticoid excessDeficiency of mineralocorticoidDecreased acid excretionIncreased base excretion  <response>: Ans. is a i.e., Primary mineralocorticoid excess Mineralocorticoid excess increases net acid excretion, this leads to metabolic alkalosis.Also knowThe plasma potassium concentration changes with serum pH.If pH decreases the excess H tends to enter the bodys cells in exchange for K and plasma increases.If the pH increases alkalosis H tends to leave cells and enters bloodstream to partly compensate for the alkalosis.In exchange for H K enters the cells and results in decreased plasma hypokalemic alkalosis.Causes of Metabolic AlkalosisExogenous HCO3 loadsAcute alkali administrationMilk-alkali syndromeEffective ECFV contraction, normotension, K deficiency, and secondary hyperreninemic hyperaldosteronismGastrointestinal originVomitingGastric aspirationCongenital chloridorrheaVillous adenomaCombined administration of sodium polystyrene sulfonate Kayexalate and aluminum hydroxide.Renal originDiureticsEdematous statesPost hypercapnic stateHypercalcemiahypoparathyroidismRecovery from lactic acidosis or ketoacidosisNonabsorbable anions including penicillin, carbenicillinMg2deficiencyK depletionBartters syndrome loss of function mutations in TALHGitelman s syndrome loss of function mutation in Na-Cl- cotransporter in DCTECFV expansion, hypertension, K deficiency, and mineralocorticoid excessHigh reninRenal artery stenosisAccelerated hypertensionRenin-secreting tumorEstrogen therapyLow reninPrimary aldosteronismAdenomaHyperplasiaCarcinomaAdrenal enzyme defectslip - Hydroxylase deficiency17a - Hydroxylase deficiencyCushings syndrome or diseaseOtherLicoriceCarbenoxoloneChewers tobaccoLydia Pinkam tabletsGain of function mutation of renal sodium channel with ECFV expansion, hypertension, K deficiency, and hyporeninemic- hypoaldosteronismA.Liddles syndromeThis is the complete list of metabolic alkalosis the list given in answer 32 is not complete. 