<prompt> All of following are anti platelet EXCEPT:AspirinClopidogrelAbciximabTranexamic acid  <response>: Ref: KDT 6th ed. pg. 608 Antiplatelet drugs interfere with platelet function and are useful in prophylaxis of thromboembolic disorders. Platelets express several glycoprotein GP receptors on their surface like GP IIaIIIb. These receptors are not exposed normally, but once exposed due to some injury they make the platelets sticky. Upon injury thromboxane A2 TXA2 and ADP produced, which expose the sticky receptors GP IIa IIIb that crosslinks the platelets, inducing aggregation. Prostacyclin PGI2 synthesized in the intima of blood vessel is an inhibitor of TXA2. Normally, a balance between PGI2 and TXA2 is required to control thrombus formation.Antiplatelet drugs and their target of inhibitionAntiplatelet drugsTarget of inhibitionAspirinInhibit TXA2Clopidogrel and TiclodipineInhibit ADP irreversibly by binding with P2Y12 receptorCangrelor, TicagrelorInhibit ADP reversiblyAbciximabTirofibanEptifibatideInhibit GP IIbIIIaFibrin is associated with clotting. Agents which lyse this fibrin is known as Fibrinolytics.Overdose of fibrinolytic agents causes results in excessive intravascular fibrinolysis resulting in bleeding.Tranexamic acid is a specific antidote for fibrinolytic agents. Antifibrinolytic drugs are: Epsilon Amino Caproic acid EACA Tranexamic acid 