<prompt> All of the following are Jnconjugated Hyperbilirubinemias EXCEPTCriggler Najjar syndrome Type 1Gilberts syndromeDubin Johnson syndromeCriggler Najjar syndrome Type 2  <response>: C Dubin Johnson syndrome Crigler-Najjar syndrome CNS is a rare autosomal recessive disorder of bilirubin metabolism. Two distinct forms have been described, as follows: type 1 and type 2. Type 1 CNS, first described in 1952 by Crigler and Najjar, is associated with neonatal unconjugated hyperbilirubinemia high levels and kernicterus. Type 2 CNS Arias syndrome, first described in 1962 by Arias, presents with a lower serum bilirubin level and responds to phenobarbital treatment. CNS is elicited by a lack or deficiency of the enzyme uridine diphosphate glycosyltransferase UGT. A markedly elevated unconjugated indirect hyperbilirubinemia is observed in inherited disorders such as Gilbert syndrome and CNS. Among the inherited unconjugated hyperbilirubinemias, Gilbert syndrome is believed to affect approximately 3-7 of the adult population. Dubin Johnson syndrome Rotor syndromes are inborn errors of metabolism which lead to conjugated hyperbilirubinemia 