 Detail: The backflow of gastric contents into the esophagus occurs in healthy people to a limited extent.1 When the refluxed material transcends the upper esophageal sphincter and enters the laryngopharynx on a chronic basis, it is termed laryngopharyngeal reflux LPR. LPR typically manifests as dysphonia, globus sensation, chronic cough, and throat irritation. Recent studies suggest that untreated LPR may be a cause of lingual tonsil hypertrophy LTH, 2,3 which itself has been implicated as a contributing factor in obstructive sleep apnea-hypopnea syndrome OSAHS.4-6 Lingual tonsil tissue LTT is present to a variable degree in most individuals and tends to regress with age. Although standardized parameters for defining LTH have yet to be established, a recent study by Friedman et al 7 demonstrated that on average subjects with LPR, OSAHS, or both diseases had significantly thicker LTT on CT imaging than patients without either disease. Del Gaudio and coworkers 2 identified a trend between increasing severity of LTH and the frequency of reflux events, while Mamede et al 3 found a positive correlation between reflux symptoms and the degree of LTH. 