<prompt> Please answer with one of the option in the bracket. Write reasoning in between . Write answer in between .Q:You are examining a 3-day-old newborn who was delivered vaginally without any complications. The newborn presents with vomiting, hyperventilation, lethargy, and seizures. Blood work demonstrates hyperammonemia, elevated glutamine levels, and decreased blood urea nitrogen. A CT scan demonstrates cerebral edema. Defects in which of the following enzymes would result in a clinical presentation similar to this infant?? A: Phenylalanine hydroxylase, B: Branched-chain ketoacid dehydrogenase, C: Homogentisate oxidase, D: Cystathionine synthase, E: Carbamoyl phosphate synthetase I,  <response>: This is a clinical vignette describing a newborn with symptoms of vomiting, hyperventilation, lethargy, and seizures along with lab findings of hyperammonemia, elevated glutamine, and decreased BUN. This presentation is consistent with a urea cycle disorder. The urea cycle is responsible for removing ammonia from the body. Defects in enzymes involved in the urea cycle can lead to hyperammonemia. Of the choices given, carbamoyl phosphate synthetase I is an enzyme involved in the urea cycle. Therefore, a defect in this enzyme would result in the clinical presentation described.E: Carbamoyl phosphate synthetase I 