<prompt> Please answer with one of the option in the bracket. Write reasoning in between . Write answer in between .Q:After hospitalization for urgent chemotherapy to treat Burkitts lymphoma, a 7-year-old boy developed paresthesias of the fingers, toes, and face. Vital signs are taken. When inflating the blood pressure cuff, the patient reports numbness and tingling of the fingers. His blood pressure is 10065 mm Hg. Respirations are 28min, pulse is 100min, and temperature is 36.2C 97.2F. He has excreted 20 mL of urine in the last 6 hours.Laboratory studies show the following:Hemoglobin 15 gdLLeukocyte count 6000mm3 with a normal differential serumK 6.5 mEqLCa 6.6 mgdLPhosphorus 5.4 mgdLHCO3 15 mEqLUric acid 12 mgdLUrea nitrogen 54 mgdLCreatinine 3.4 mgdLArterial blood gas analysis on room air:pH 7.30PCO2 30 mm HgO2 saturation 95Which of the following is the most likely cause of this patients renal condition?? A: Deposition of calcium phosphate in the kidney, B: Direct tubular toxicity through filtered light chains, C: Intense renal vasoconstriction and volume depletion, D: Pigment-induced nephropathy, E: Precipitation of uric acid in renal tubulestumor lysis syndrome,  <response>: This is a clinical case about a 7-year-old boy who recently underwent chemotherapy for Burkitts lymphoma and now has signs and symptoms of acute kidney injury, including paresthesias, decreased urine output, and abnormal laboratory results showing elevated serum creatinine, BUN, potassium, uric acid, and low bicarbonate. The key findings pointing to the likely diagnosis are:- Recent chemotherapy for a tumor Burkitts lymphoma that has high proliferation rate and turnover - Elevated uric acid level- Metabolic acidosis with low serum bicarbonate - HyperkalemiaThese findings are most consistent with tumor lysis syndrome, where rapid tumor cell breakdown leads to release of intracellular contents uric acid, potassium, phosphorus that precipitate in the renal tubules and cause acute kidney injury.The other answer choices may play a role but are less likely to be the primary cause of the acute kidney injury in this clinical scenario.E: Precipitation of uric acid in renal tubulestumor lysis syndrome 