<prompt> Please answer with one of the option in the bracket. Write reasoning in between . Write answer in between .Q:A 4-day-old boy is brought to the physician because of somnolence, poor feeding, and vomiting after his first few breast feedings. He appears lethargic. His respiratory rate is 73min. Serum ammonia is markedly increased. Genetic analysis shows deficiency in N-acetylglutamate synthase. The activity of which of the following enzymes is most likely directly affected by this genetic defect?? A: Carbamoyl phosphate synthetase I, B: Ornithine translocase, C: Argininosuccinate synthetase, D: Argininosuccinase, E: Arginase,  <response>: This is a question about an enzyme deficiency leading to hyperammonemia in a newborn. The key information in the stem is that the patient has a genetic deficiency in N-acetylglutamate synthase. N-acetylglutamate is an allosteric activator of carbamoyl phosphate synthetase I, which is the rate-limiting enzyme in the urea cycle. Therefore, a deficiency in N-acetylglutamate synthase would directly affect the activity of carbamoyl phosphate synthetase I.A: Carbamoyl phosphate synthetase I 