<prompt> Please answer with one of the option in the bracket. Write reasoning in between . Write answer in between .Q:A 6-year-old boy is brought to the physician because of a 2-week history of frequent episodes of unresponsiveness. During these episodes, he stares blankly, rhythmically nods his head, and does not respond to verbal stimulation for several seconds. Hyperventilation for 30 seconds precipitates an episode of unresponsiveness and head nodding that lasts for 7 seconds. He regains consciousness immediately afterward. An electroencephalogram shows 3-Hz spikes and waves. Which of the following best describes the mechanism of action of the most appropriate pharmacotherapy for this patients condition?? A: Increased frequency of GABAA channel opening, B: Blockade of thalamic T-type calcium channels, C: Irreversible inhibition of GABA transaminase, D: Increased duration of GABAA channel opening, E: Inhibition of GABA reuptake into presynaptic neurons,  <response>: This is a clinical vignette describing a 6-year-old boy with episodes of unresponsiveness, staring blankly, head nodding, and no response to verbal stimulation lasting several seconds. These episodes can be precipitated by hyperventilation. An EEG shows 3-Hz spike-wave discharges, consistent with absence seizures. The question asks about the mechanism of action of the most appropriate pharmacotherapy for this patient. Based on the clinical presentation of typical absence seizures and the EEG findings, this patient most likely has childhood absence epilepsy. The first-line treatment for absence seizures is ethosuximide, which blocks T-type calcium channels in the thalamus.B: Blockade of thalamic T-type calcium channels 