<prompt> Please answer with one of the option in the bracket. Write reasoning in between . Write answer in between .Q:A 12-month-old child passed away after suffering from craniofacial abnormalities, neurologic dysfunction, and hepatomegaly. Analysis of the childs blood plasma shows an increase in very long chain fatty acids. The cellular analysis demonstrates dysfunction of an organelle responsible for the breakdown of these fatty acids within the cell. Postmortem, the child is diagnosed with Zellweger syndrome. The family is informed about the autosomal recessive inheritance pattern of the disease and their carrier status. Which of the following processes is deficient in the dysfunctional organelle in this disease?? A: Beta-oxidation, B: Transcription, C: Translation, D: Ubiquitination, E: Phosphorylation,  <response>: This is a question about Zellweger syndrome, which is caused by a deficiency in peroxisomes. The key details in the question stem are:- Child had craniofacial abnormalities, neurologic dysfunction, and hepatomegaly - Increased very long chain fatty acids in blood- Dysfunction of an organelle responsible for breakdown of fatty acids- Diagnosed with Zellweger syndrome, an autosomal recessive disorderThis points to a defect in peroxisomes, which are organelles that break down very long chain fatty acids via beta-oxidation. The question asks which process is deficient in the dysfunctional organelle.A: Beta-oxidation 