<prompt> Please answer with one of the option in the bracket. Write reasoning in between . Write answer in between .Q:A newborn boy develops projectile vomiting 48 hours after delivery. He is found to be lethargic, with poor muscle tone, and is hyperventilating. Within hours, he suffers important neurological deterioration, leading to seizures, coma, and, ultimately, death. An autopsy is performed and the pathology team makes a diagnosis of a rare genetic disorder that leads to low levels of N-acetylglutamate. Which of the following enzymes would be secondarily affected by this process?? A: Carbamoyl phosphate synthetase I, B: Ornithine transcarbamylase, C: Arginase, D: Argininosuccinate lyase, E: Argininosuccinate synthetase,  <response>: This is a question about a newborn with symptoms of lethargy, poor muscle tone, hyperventilation, seizures, coma and death, who is diagnosed with a rare genetic disorder leading to low levels of N-acetylglutamate. The question asks which enzyme would be secondarily affected by this process. Based on the symptoms and diagnosis, this appears to be a urea cycle disorder, specifically a deficiency in N-acetylglutamate synthase. N-acetylglutamate is an allosteric activator of carbamoyl phosphate synthetase I, the first enzyme in the urea cycle. Therefore, a deficiency in N-acetylglutamate would secondarily affect carbamoyl phosphate synthetase I activity.Of the choices given, carbamoyl phosphate synthetase I is the enzyme that would be most directly and severely affected by low levels of its allosteric activator N-acetylglutamate. The other enzymes listed are part of the urea cycle but would not be as directly impacted.A: Carbamoyl phosphate synthetase I 