<prompt> Please answer with one of the option in the bracket. Write reasoning in between . Write answer in between .Q:A 46-year-old woman presents with palpitations, tremors, and anxiety. She says these symptoms have been present ever since a recent change in her diabetic medication. The most recent time she felt these symptoms, her blood glucose level was 65 mgdL, and she felt better after eating a cookie. Which of the following is the mechanism of action of the drug most likely to have caused this patients symptoms?? A: Inhibitor of dipeptidyl peptidase DPP-IV, B: Block reabsorption of glucose in proximal convoluted tubule PCT, C: Inhibition of α-glucosidase, D: Blocking of the ATP-sensitive K channels, E: Decreased hepatic gluconeogenesis,  <response>: This is a question asking about the mechanism of action of a diabetic medication that is causing hypoglycemic symptoms like palpitations, tremors, and anxiety in a patient. The key information is that the patients blood glucose dropped to 65 mgdL and she felt better after eating something sugary. This indicates that the medication is causing hypoglycemia, or low blood sugar. Going through the choices, inhibitors of DPP-IV, alpha-glucosidase, and drugs that block glucose reabsorption in the PCT would not cause hypoglycemia. Decreased hepatic gluconeogenesis may cause hypoglycemia but is not a common mechanism for diabetic medications. The answer that matches hypoglycemia as a side effect is blocking ATP-sensitive potassium channels. This is the mechanism of action of sulfonylureas, a class of oral diabetic medications that can cause hypoglycemia in some patients.D: Blocking of the ATP-sensitive K channels 