<prompt> Please answer with one of the option in the bracket. Write reasoning in between . Write answer in between .Q:A medical student is spending his research year studying the physiology of cholesterol transport within the body. Specifically, he wants to examine how high density lipoprotein HDL particles are able to give other lipoproteins the ability to hydrolyse triglycerides into free fatty acids. He labels all the proteins on HDL particles with a tracer dye and finds that some of them are transferred onto very low density lipoprotein VLDL particles after the 2 are incubated together. Furthermore, he finds that only VLDL particles with transferred proteins are able to catalyze triglyceride hydrolysis. Which of the following components were most likely transferred from HDL to VLDL particles to enable this reaction?? A: Apo-A1, B: ApoB-100, C: ApoC-II, D: ApoE, E: Lipoprotein lipase,  <response>: This is a question about the physiology of cholesterol transport and specifically about how HDL particles enable VLDL particles to hydrolyze triglycerides. The key points in the question stem are:- The medical student labeled HDL proteins with a tracer dye and found some were transferred to VLDL particles after incubation- Only VLDL particles with the transferred proteins could catalyze triglyceride hydrolysis- The question asks which component transferred from HDL to VLDL enabled this triglyceride hydrolysis reactionBased on this information, we can narrow down the answer choices to components that could be transferred from HDL to VLDL and enable triglyceride hydrolysis. ApoC-II is a likely answer since it is an HDL protein that acts as a cofactor for lipoprotein lipase, the enzyme that catalyzes triglyceride hydrolysis. ApoE and lipoprotein lipase are less likely since they are not intrinsic HDL proteins. ApoA-I and ApoB-100 are components of HDL and VLDL respectively, but do not directly enable triglyceride hydrolysis.C: ApoC-II 