<prompt> Please answer with one of the option in the bracket. Write reasoning in between . Write answer in between .Q:A 47-year-old female with a history of mild asthma, type II diabetes, hypertension, and hyperlipidemia presents to clinic complaining of swelling in her lips Image A. She has had no changes to her medications within the past two years. Vital signs are stable. Physical exam is notable for significant erythema around and swelling of the lips. The remainder of her exam is unremarkable. What is the mechanism of action of the drug that has caused her current symptoms?? A: Inhibition of angiotensin-converting enzyme, B: Inhibition of HMG-CoA reductase, C: Stimulation of the Beta 2 receptor, D: Inhibition of the NaKCl triple transporter of the thick ascending limb, E: Inhibition of voltage-dependent L-type calcium channels,  <response>: This is a clinical vignette describing a patient with a history of asthma, diabetes, hypertension, and high cholesterol who is presenting with lip swelling. The question is asking about the mechanism of action of the medication that is likely causing her lip swelling. Based on the history provided, the main medications she is likely taking are:- A beta agonist inhaler for asthma e.g. albuterol- An ACE inhibitor or ARB for hypertension e.g. lisinopril, losartan - A statin for hyperlipidemia e.g. atorvastatin- Potentially a sulfonylurea or metformin for diabetesLip swelling is a classic side effect of ACE inhibitors like lisinopril due to buildup of bradykinin. This makes choice A the most likely answer.The other medications do not cause lip swelling through their mechanisms of action. Choice B involves statins which lower cholesterol. Choice C involves beta agonists which relax bronchial smooth muscle. Choice D involves diuretics which reduce sodium reabsorption. Choice E involves calcium channel blockers which relax vascular smooth muscle.A: Inhibition of angiotensin-converting enzyme 