<prompt> Please answer with one of the option in the bracket. Write reasoning in between . Write answer in between .Q:Maturity Onset Diabetes of the Young MODY type 2 is a consequence of a defective pancreatic enzyme, which normally acts as a glucose sensor, resulting in a mild hyperglycemia. The hyperglycemia is especially exacerbated during pregnancy. Which of the following pathways is controlled by this enzyme?? A: Glucose -- glucose-6-phosphate, B: Glucose-6-phosphate -- fructose-6-phosphate, C: Fructose-6-phosphate -- fructose-1,6-biphosphate, D: Glyceraldehyde-3-phosphate -- 1,3-bisphosphoglycerate, E: Phosphoenolpyruvate -- pyruvate,  <response>: This question is asking about the specific enzyme involved in Maturity Onset Diabetes of the Young MODY type 2. MODY type 2 is caused by mutations in the glucokinase GCK gene, which encodes an enzyme that acts as a glucose sensor and regulates insulin secretion. GCK catalyzes the conversion of glucose to glucose-6-phosphate, which is the first step of glycolysis. Therefore, the defective GCK enzyme would affect the Glucose -- glucose-6-phosphate pathway.A: Glucose -- glucose-6-phosphate 