<prompt> Please answer with one of the option in the bracket. Write reasoning in between . Write answer in between .Q:A 27-year-old woman presented to the clinic with recurrent abdominal swelling and stunted growth relative to her siblings. She has a history of multiple blood transfusions in her childhood. She has a family history of jaundice in her father who was operated on for multiple gallbladder stones. The physical examination reveals a pale, icteric, small and short-statured young lady. On abdominal examination, the spleen was enlarged by 6 cm below the right costal margin, but the liver was not palpable. The ultrasound of the abdomen reveals multiple gallbladder stones. The laboratory test results are as follows:Hb 9 gdLHct 27WBC 6,200mm3Platelets 200,000mm3MCV 75 um3MCHC 37 gmdLReticulocytes 6.5A peripheral blood smear is presented in the image. The direct Coombs test was negative. The osmotic fragility test was increased. What is the most likely cause of her condition?? A: Hereditary spherocytosis, B: Blood loss, C: Aplastic anemia, D: Vitamin B12 deficiency, E: Anemia of chronic disease,  <response>: This is a clinical case of a 27-year-old woman with a history of recurrent abdominal swelling, stunted growth, multiple blood transfusions in childhood, and family history of jaundice in her father who had gallbladder stones. The key findings on physical exam are pallor, icterus, splenomegaly, and short stature. Lab tests show anemia low Hb and Hct, normal WBC, thrombocytopenia, increased MCV, normal MCHC, increased reticulocytes, and increased osmotic fragility. The peripheral blood smear is provided. The recurrent abdominal swelling, splenomegaly, family history of gallstones, and increased osmotic fragility point towards a hemolytic anemia. The increased MCV and reticulocytosis indicate a regenerative anemia. The normal MCHC rules out iron deficiency. The negative Coombs test rules out an autoimmune hemolytic anemia. Of the choices given, hereditary spherocytosis best fits the clinical and laboratory findings in this patient.A: Hereditary spherocytosis 