<prompt> Please answer with one of the option in the bracket. Write reasoning in between . Write answer in between .Q:A 13-year-old girl is brought to the physician by her mother because of a 1-month history of abnormal movements of her muscles that she cannot control. She has a younger brother with cognitive disabilities and epilepsy. Examination shows frequent, brief, involuntary contractions of the muscle groups of the upper arms, legs, and face that can be triggered by touch. An EEG shows generalized epileptiform activity. A trichrome stain of a skeletal muscle biopsy specimen shows muscle fibers with peripheral red inclusions that disrupt the normal fiber contour. Which of the following is the most likely underlying mechanism of the patients symptoms?? A: CTG trinucleotide repeat expansion, B: Mutation of the methyl-CpG binding protein 2 gene, C: Defective oxidative phosphorylation, D: Autoimmune endomysial destruction, E: Truncated dystrophin protein,  <response>: This is a clinical vignette describing a 13-year-old girl with a 1-month history of involuntary muscle contractions myoclonus that can be triggered by touch stimulus-sensitive myoclonus. She has a brother with cognitive disabilities and epilepsy, suggesting a possible geneticfamilial cause. Her EEG shows epileptiform discharges, and muscle biopsy shows red rimmed vacuoles, which are characteristic of certain muscular dystrophies. The key findings are:- Stimulus-sensitive myoclonus - Family history of neurological disorders- EEG abnormalities - Red rimmed vacuoles on muscle biopsyThis constellation of findings is most consistent with myoclonus epilepsy and ragged red fibers MERRF, a mitochondrial disorder caused by defective oxidative phosphorylation.C: Defective oxidative phosphorylation 