<prompt> Please answer with one of the option in the bracket. Write reasoning in between . Write answer in between .Q:A 10-year-old boy is brought to the emergency department by his mother due to frequent vomiting, abdominal pain, and weakness. Over the last 5 days, has been noted to have polydipsia and polyuria. Family history is irrelevant. His temperature is 37.1C 98.7F, blood pressure is 10070 mm Hg, and pulse is 110min. Physical examination reveals no response to verbal commands, sunken eyes, poor skin turgor, and rapid deep respirations. Laboratory results are shown:Random plasma glucose 420 mgdLSerum beta-hydroxybutyrate elevatedFasting C-peptide undetectableAntiglutamic acid decarboxylase GAD antibodies positiveThis patients condition occurs as a result of which of the following?? A: Insulin resistance, B: Immune-mediated destruction of pancreatic beta cells, C: Starvation, D: Salicylate poisoning, E: Defective synthesis or release of arginine vasopressin,  <response>: Based on the information provided in the question stem, this 10-year-old boy is presenting with symptoms of polydipsia, polyuria, vomiting, abdominal pain, weakness, sunken eyes, poor skin turgor, rapid deep respirations, elevated random plasma glucose, elevated serum beta-hydroxybutyrate, undetectable fasting C-peptide, and positive antiglutamic acid decarboxylase GAD antibodies. The key findings here are the elevated blood glucose, low C-peptide, and positive GAD antibodies. C-peptide is produced when proinsulin is split into insulin and C-peptide, so low levels indicate insufficient insulin production. GAD antibodies are markers of autoimmune destruction of pancreatic beta cells which produce insulin. Together, these findings point to an autoimmune destruction of the pancreatic beta cells leading to insulin deficiency and hyperglycemia. The other answer choices do not fit with the clinical and laboratory picture.B: Immune-mediated destruction of pancreatic beta cells 