<prompt> Please answer with one of the option in the bracket. Write reasoning in between . Write answer in between .Q:A 3-year-old boy is seen in clinic. He was born at home without perinatal care. He was apparently normal at birth, but later developed failure to thrive and developmental delay. He also has a history of cataracts. His older brother had a myocardial infarction at the age of 18 and is rather lanky and tall in appearance. Laboratory testing of his urine showed an increase in the level of an amino acid. What is the most likely mechanism responsible for this boys pathology?? A: Hereditary defect of renal amino acid transporter, B: Cystathionine synthase deficiency, C: Decreased in phenylalanine hydroxylase, D: Deficiency of homogentisic acid oxidase, E: Inability to degrade branched chain amino acids,  <response>: This is a clinical vignette describing a 3-year-old boy with failure to thrive, developmental delay, cataracts, and increased urinary amino acids. His older brother had a myocardial infarction at 18 and is tall and lanky. This history is classic for homocystinuria, an inborn error of metabolism caused by cystathionine beta-synthase deficiency. The inability to convert homocysteine to cystathionine leads to accumulation of homocysteine and methionine. Elevated homocysteine causes cardiovascular disease. The increased methionine is excreted in the urine as amino acids.B: Cystathionine synthase deficiency 