<prompt> Please answer with one of the option in the bracket. Write reasoning in between . Write answer in between .Q:An investigator studying influenza virus variability isolates several distinct influenza virus strains from the respiratory secretions of a study subject. Mass spectrometry analysis of one strain shows that it expresses neuraminidase on its surface. Subsequent sequencing of this strain shows that its genome lacks the neuraminidase gene. Which of the following is the most likely explanation for this finding?? A: Transduction, B: Reassortment, C: Recombination, D: Phenotypic mixing, E: Complementation,  <response>: This question is asking about the mechanism that would allow an influenza virus strain to express a protein neuraminidase even though its genome lacks the gene encoding that protein. The key finding is that the virus expresses a protein that its own genome does not encode. This suggests that the protein was acquired from a different virus strain through some process of genetic exchange.Transduction involves transfer of bacterial DNA by a bacteriophage and does not apply here. Reassortment involves mixing of genome segments between different influenza virus strains during coinfection. This could potentially lead to expression of a protein not encoded in that strains own genome.Recombination also allows genetic exchange between viruses but at smaller scales by breakage and rejoining of DNA strands. This could also potentially lead to the observed result.Phenotypic mixing refers to expression of proteins from genetically distinct viruses during coinfection of a cell. This seems the most likely explanation here.Complementation involves a defective virus borrowing a function from a coinfecting virus. This does not seem to fit the scenario described.D: Phenotypic mixing 