<prompt> Please answer with one of the option in the bracket. Write reasoning in between . Write answer in between .Q:A 35-year-old woman presents to the emergency department with severe nausea and diarrhea. One day prior to presentation, she went to a new seafood restaurant known for serving exotic fish. For the past day she experienced nausea, diarrhea, weakness, and a tingling sensation in her extremities. In the emergency department, her temperature is 100.3F 37.9C, blood pressure is 9560 mmHg, pulse is 105min, and respirations are 20min. On physical examination, she appears fatigued and has 1 Achilles and patellar reflexes. Which of the following is the mechanism of action of the compound most likely responsible for this patients clinical presentation?? A: Promotes depolarization of Na channels, B: Prevents depolarization of Na channels, C: Increases synthesis of histamine, D: Superantigen that activates T-cells, E: Permanent Gs activation,  <response>: This is a clinical vignette describing a 35-year-old woman with acute onset nausea, diarrhea, weakness and tingling after eating at a new seafood restaurant. The symptoms, timeline, physical exam findings of low blood pressure, tachycardia, hyporeflexia, and low grade fever are consistent with food poisoning. The mechanism of action question is asking about the toxin most likely responsible. Based on the acute gastrointestinal and neurological symptoms after seafood ingestion, this is most likely due to ciguatoxin, which binds to voltage-gated sodium channels causing them to remain open, leading to neuronal excitability and symptoms.B: Prevents depolarization of Na channels 