<prompt> Please answer with one of the option in the bracket. Write reasoning in between . Write answer in between .Q:During an experiment conducted to alter the infectivity of common viruses that affect humans, an investigator successfully increases the host range of human immunodeficiency virus HIV. The new strain of the virus can infect fibroblast-like cells in addition to the usual target of HIV. Which of the following is the most likely explanation for the increase in the host range of the virus?? A: Mutation of the gene coding for viral surface glycoproteins, B: Point mutations in the hemagglutinin gene, C: Increased rate of budding out of host cells, D: Reassortment of genetic material between segments of two viruses, E: Excessive activity of viral RNA polymerase,  <response>: This is a question about virology and how viruses can evolve to infect new host cells. The key is understanding that HIV specifically infects immune cells like T cells and macrophages, but not other cell types like fibroblasts. For HIV to gain the ability to infect fibroblasts, its surface proteins that mediate cell entry likely need to change via mutation. Choice A about viral surface glycoprotein mutation is therefore the most plausible explanation. Choices B-E describe mechanisms unrelated to changing viral tropism or host range.A: Mutation of the gene coding for viral surface glycoproteins 