<prompt> Please answer with one of the option in the bracket. Write reasoning in between . Write answer in between .Q:A 21-year-old G1P0 woman presents to the labor and delivery ward at 39 weeks gestation for elective induction of labor. She requests a labor epidural. An epidural catheter is secured at the L4-L5 space. She exhibits no hemodynamic reaction to lidocaine 1.5 with epinephrine 1:200,000. A continuous infusion of bupivacaine 0.0625 is started. After 5 minutes, the nurse informs the anesthesiologist that the patient is hypotensive to 8050 mmHg with a heart rate increase from 90 bpm to 120 bpm. The patient is asymptomatic and fetal heart rate has not changed significantly from baseline. She says that her legs feel heavy but is still able to move them. What is the most likely cause of the hemodynamic change?? A: Bainbridge reflex, B: Intrathecal infiltration of local anesthetic, C: Local anesthetic systemic toxicity, D: Spinal anesthesia, E: Sympathetic blockade,  <response>: This is a question about the side effects of epidural anesthesia in a pregnant woman. The key details are that the patient received an epidural catheter at L4-L5, exhibited no reaction to the test dose of lidocaine with epinephrine, and then developed hypotension and tachycardia 5 minutes after starting a continuous infusion of dilute bupivacaine. She is asymptomatic but has some motor block. This presentation is most consistent with sympathetic blockade from the epidural, which can cause hypotension and reflex tachycardia. Intrathecal infiltration would cause more significant motor block. Local anesthetic systemic toxicity would also cause more CNS and cardiovascular effects. Bainbridge reflex and spinal anesthesia do not fit with the clinical scenario described.E: Sympathetic blockade 