 Introduction: Multiple sclerosis is a chronic inflammatory demyelinating disease of the central nervous system that predominantly affects young adults 1. Although its etiology has not yet been elucidated, evidence points to an autoimmune pathogenesis where it is thought that self-reactive and myelin-specific cluster of differentiation four and eight CD4and CD8 T cells, play an important role by reacting and destroying the myelin sheath 2. No plasma biomarkers to determine disease activity by routine clinical measurements, the existence a pattern of pro-inflammatory cytokines has been described, with range increased Interferon IFN, Tumoral Necrosis Factor alpha TNFa and Interleukin 12 IL-12, as well as a decline in Interleukin10 IL10 and Interleukin 4 IL4 anti-inflammatory cytokines 3. To date, the most common treatment for MS is Interferon beta immunomodulation, although its cost effectiveness has been questioned4. 